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namic, activitydependent translocation in active neurons could i) be regulated by interacting structural or signaling proteins and/or ii) serve to recruit together these proteins inside the CaMKII scaffolds at strategic web pages such as the synapse or intra-somatic elements. One particular probable regulator of CaMKII action will be the RGK household of Ras-related small GTPases, which incorporat
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namic, activitydependent translocation in active neurons could i) be regulated by interacting structural or signaling proteins and/or ii) serve to recruit together these proteins within the CaMKII scaffolds at strategic web sites like the synapse or intra-somatic components. One attainable regulator of CaMKII action could be the RGK loved ones of Ras-related compact GTPases, which incorporates the
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een proven by the deleterious consequences of CFTR mutations that result in Cystic Fibrosis, an autosomal genetic illness. CF phenotype is dominated by alterations in epithelial secretions. These abnormal secretions are connected to CFTR defects, inside a direct or indirect manner. The loss of interactions in between CFTR and also other ion transporters have important consequences: the poor hydrat